LCN2 and acute kidney injury: Since circulating hemeproteins are known to induce kidney injury, intraoperative lipid peroxidation independently predicts postoperative AKI,[7] and acetaminophen inhibited hemeprotein-mediated lipid peroxidation and preserved kidney function in an animal model of rhabdomyolysis, we performed an exploratory analysis of postoperative AKI.[5] Acetaminophen did not affect postoperative creatinine concentrations, postoperative urinary NGAL concentrations, or the incidence of AKI.