Merkel et al. [29] demonstrated that members of the miR-17-92 cluster, which have been associated with inhibition of apoptosis, promotion of proliferation and induction of tumor angiogenesis are highly expressed in ALK+ ALCL, whereas miR-155, which is involved in the immune response and has oncogenic potential, was expressed at higher levels in ALK- ALCL. This evidence concerns the gene ALK and neoplasm.