SFV-F3A–infected cells had a significantly higher number of SG-positive cells than wt SFV–infected cultures (18.4%), and this proportion was increased to 49.2% by the Pat A treatment, indicating that in the absence of nsP3/G3BP interaction, infection-induced SGs persist longer and additional SGs can be stress-induced in SFV-F3A-infected cells. The gene discussed is SH2D3C; the disease is infection.