The most severe sequel, the hemolytic uremic syndrome (HUS), results from shiga toxins (Stxs) produced by the bacteria in the intestine that act systemically on sensitive cells in the kidneys, brain, and other organs.1 Although most EHEC strains produce Stxs, those produced by EHEC O157:H7 are particularly virulent and are responsible for the majority of HUS cases of bacterial etiology worldwide.1, 2 In addition to shiga toxins, the eaeA gene that encodes for intimin and the hly gene that encodes for hemolysin are other main virulence factors.3,4. This evidence concerns the gene KRCC1 and hemolytic-uremic syndrome.