To support the hypothesis that IFIT3 expression is factually accompanied by an “inflammatory status” in pancreatic cancer cells, we examined the secretion of the inflammatory cytokines IL-6, TNF-α, IL-1ß, IL-2, and IL-10 with respect to the expression of IFIT3 and SOX9 in respective culture supernatants. The gene discussed is IL1B; the disease is pancreatic neoplasm.