HLA-A and classic Hodgkin lymphoma: Moutsianas et al. (2011) analysed HLA‐A, C, B, DRB1, DQA1 and DQB1 alleles imputed from their GWAS data, but did not stratify patients by EBV status.27 In unconditional analyses, an increased risk of cHL was associated with DRB1*15:01, DQB1*06:02 and DQB1*03:03 and a decreased risk with DRB1*07:01 and DQA1*02:01; however, the effect of rs6903608 could not be explained by these alleles and they concluded that rs6903608, DQA1*02:01 and the DPB1 SNP rs2281389 were the main independent contributors to disease risk.