Furthermore, down-regulated genes associated with high let-7a-2–3p expression included known oncogene JDP2 [23], and several genes in the family of leukocyte immunoglobulin-like receptor (LILRA5/6, LILRB2/3), which might contribute to the adverse prognosis of CN-AML. The gene discussed is JDP2; the disease is acute myeloid leukemia.