AGT and Parkinson disease: Although the phenotypic identity of the neural cells examined was not identified by this 1996 report by Ge and Barnes [21], they did report that relative to that present in age-, sex-, and post-mortem interval-matched neurologically normal individuals, there is a marked reduction in AngII binding to AT1 in the caudate, putamen, and substantia nigra in brains of Parkinson patients.