Increased fibrosis is common in DCM patients (Assomull et al., 2006; Herpel et al., 2006) and the cardiac STAT3 KO mouse model of PPCM (Ricke-Hoch et al., 2014), however, there are limited and contradictory reports about fibrosis in PPCM patients (Kawano et al., 2008; Mouquet et al., 2008; Leurent et al., 2009; Ntusi and Chin, 2009). The gene discussed is STAT3; the disease is familial dilated cardiomyopathy.