Effects of luteolin administration on the expressions of cMet, p-cMet, Akt, p-Akt, ERK, p-ERK, MMP9 and Ki-67 in PDTX models of gastric cancer cMet-overexpression in gastric cancer is accompanied by abnormal activation of cMet signaling that contributes to tumor survival, growth and metastasis. This evidence concerns the gene AKT1 and neoplasm.