However, considering the high prevalence of other comorbidities associated with elevated hs-cTnT levels among our cohort, including age, heart failure, diabetes, ischemic heart disease, hypertension and renal failure it is conceivable that structural changes such as left ventricular hypertrophy and systolic dysfunction were highly prevalent among our cohort. The gene discussed is TNNT2; the disease is left ventricular hypertrophy.