Irrespective of these differences, the list of candidate genes we observed is most consistent with the one reported by Kapoor et al., which concluded that YAP cooperates with E2F1 in Kras induced pancreatic adenocarcinoma (PDAC) by modulating a large subset of E2F1 targets including Cdc6, Cdk1, Mcm complex components and Rad51 [20]. This evidence concerns the gene CDK1 and pancreatic adenocarcinoma.