NFKB1 and lip and oral cavity carcinoma: Since we have previously shown that γ-tocotrienol suppressed the activation of NF-κB induced by a chemotherapeutic agent (docetaxel) in oral cancer cells (25), mechanism involved in the cell survival of RT7 by γ-tocotrienol might involve regulation of the balance between NF-κB-related pro-apoptotic and anti-apoptotic genes (3).