Under IBA + Kin treatment, in contrast to lax3 and the wt, the aux1-21 AR response remained almost totally constituted by ANERs, but the density increased in comparison with the HF treatment, as in lax3 and the wt, remaining, however, less than the half that of the latter genotypes (Fig. 3B and inset). The gene discussed is AR; the disease is hydrops fetalis.