Recently, we have also investigated TNFα polymorphisms (−308 G/A, −863 C/A, and −857 C/T) and their expression in GBS patients and found that −308 G/A and −857 C/T polymorphisms with increased TNFα levels may predict susceptibility to axonal subtypes of GBS [58]. This evidence concerns the gene TNF and Guillain-Barre syndrome.