All detected abomasal ulcers in examined buffaloes were classified as type I, non-perforating erosions of abomasal mucosa, which are mucosal defects and do not penetrate the deeper layers of abomasum.5 It seems that little penetration of type I abomasal ulcer to the abomasal layers with no consequences, such as peritonitis (which occurs in type III and IV abomasal ulcer) or bleeding (which occurs in type II abomasal ulcer), causes a negligible effect on the blood serum Hp and SAA concentrations. The gene discussed is HP; the disease is peritonitis.