Compared with baseline, at the end of 3 months, ABCA1-mediated cholesterol efflux to apoA-1 was significantly increased in CAD-abs (8.14 ± 1.61% vs. 11.47 ± 3.61%; p = 0.004) but not in CAD-smo (8.16 ± 1.55% vs. 8.62 ± 1.49%; p = 0.473) (Fig. 3E). This evidence concerns the gene ABCA1 and coronary artery disorder.