Electroencephalogram recordings revealed that α1−/−;α1G-/- mice double-null for the GABA receptor α1 subunit (Gabra1) and CaV3.1 T-type Ca2+ channels (Cacna1g), in which the tremor caused by the absence of Gabra1 is potentiated by the absence of Cacna1g, showed a coherent TRCP that exhibited an onset that preceded the initiation of behavioral tremor by 3 ms. The gene discussed is CACNA1G; the disease is Tremor.