Either knockout of the Cacna1g gene encoding the CaV3.1 channel α1G subunit (α1G−/− mice) or pharmacological blockage of T-type Ca2+ channels in the inferior olive abolishes 10–15 Hz harmaline-induced tremor and the pathological oscillation [8], suggesting that activity of T-type calcium channel is critical for generating harmaline tremor. Here, CACNA1G is linked to Tremor.