When the neutrophils were isolated from tumor-bearing mice and pG/pI6-mice, the expression of Ccl17 gene was induced by T-sMs in the neutrophils from C-PC, but not the neutrophils from I-PC (Figure 1B), suggesting that the phenotype of the neutrophils might be switched to antitumorigenic in inflammatory environment, since Ccl17 is expressed in the neutrophils with a protumorigenic phenotype but not those with antitumorigenic phenotype [12, 23, 24]. Here, CCL17 is linked to neoplasm.