In patients with PD, there was a significant effect of MAPT haplotype with less activation in H1 homozygotes than H2 carriers bilaterally in the hippocampus, inferior temporal lobe, fusiform, and parahippocampal gyri at the cluster-corrected threshold (p < 0.05 FWE corrected; Fig. 3F; Table 2). The gene discussed is MAPT; the disease is Parkinson disease.