Recent reports indicate the important role of c-Myc not only on tumor growth but also on the wound-healing response.[12] Several preclinical studies have reported the effects of inhibiting c-Myc on the treatment of lung fibrosis, [30] on the stenosis of coronary arteries, [31] and on malignant tumors.[17] c-Myc can be considered an important molecular target to prevent tracheal stenosis due to the unnecessary cell proliferation in the injured airway mucosa. Here, MYC is linked to neoplasm.