Recent reports indicate the important role of c-Myc not only on tumor growth but also on the wound-healing response.[12] Several preclinical studies have reported the effects of inhibiting c-Myc on the treatment of lung fibrosis, [30] on the stenosis of coronary arteries, [31] and on malignant tumors.[17] c-Myc can be considered an important molecular target to prevent tracheal stenosis due to the unnecessary cell proliferation in the injured airway mucosa. This evidence concerns the gene MYC and cancer.