HTT and Huntington disease: Previously, we detected an abnormal activation of store-operated calciumchannels in human SK-N-SH neuroblastoma cells, expressing the full-lengthmutant huntingtin protein for the modeling of HD[13].In addition, we demonstrated that the store-operatedcalcium entry can be considered as a potential target for therapeuticintervention in the development of new approaches for treating HD.