LPS are mainly decomposed by neutrophils, eosinophils and basophils as well as liver Kupffer cells, which release then IL-1, IL-6, TNF-α and other inflammatory cytokines that attract leukocytes and trigger further inflammatory processes eventually leading to a systemic inflammatory response syndrome and local tissue injuries particularly of internal organs [9]–[11]. The gene discussed is TNF; the disease is systemic inflammatory response syndrome.