In our study, the expression level of KAP1 was higher in ovarian cancer samples than non-tumor ovarian tissues (Figure 1A–D), and the positive rates of KAP1 expression were significantly higher in ovarian epithelial cancer (55.7%) and borderline tumor (20.0%) than in normal ovarian tissue (5.0%) (all p < 0.01), which suggested that KAP1 may play an important role in ovarian cancer genesis. This evidence concerns the gene TRIM28 and ovarian cancer.