Based on our current findings it is possible that a combination of TSLP/CCL11/eotaxin-1 can serve as a better marker of the TSLP-driven eosinophilic airway pro-asthmatic responses, and perhaps it might provide useful information for risk stratification and evaluation of treatment strategies for viral-induced asthma exacerbations. This evidence concerns the gene CCL11 and asthma.