Previous studies have highlighted the heterogeneity of distinct EMT programs and the plethora of potential targets, for example, FGFR (57), Axl (96), PDGFR (97), JAK-STAT (98, 99), FAK (100), contributing to the viability of mesenchymal-like tumor cells in a model specific manner. This evidence concerns the gene PDGFRB and neoplasm.