In their subsequent studies, Kim et al. [37] stated that the agonist of P2X7 receptor (BzATP; 2',3'-O-(benzoyl-4-benzoyl)-ATP) induces overexpression of TNF-α in neurons of the region CA3 of hippocampus and as a consequence, TNF-α induces a route preventing neuronal death, and hence attenuates harmful effects of status epilepticus. This evidence concerns the gene P2RX7 and status epilepticus.