To substantiate these mechanisms, experimental animal models were described for Hashimoto's thyroiditis [55], experimental acute encephalitis (EAC) [56], and CVB3 myocarditis [57], whereby lesions were produced, respectively, by injections of thyroglobulin, myelin basic proteins (MBP), or CVB3 antigens, each mixed with adjuvant [56]–[57]. The gene discussed is MBP; the disease is myocarditis.