Firstly, the subset prevalence of γδTc in the original tumor was not reported and thus (regulatory) Vδ2Tc may have comprised the majority of tumor-derived cell suspensions at the outset but may have been subsequently eliminated by high levels of IL-2 in the culturing process, since Vδ2Tc are known to be susceptible to activation-induced cell death (34–36). Here, IL2 is linked to neoplasm.