DcR3 is known to be also a soluble receptor of LIGHT [15] (homologous to Lymphotoxins exhibiting Inducible expression and competing with herpes simplex virus Glycoprotein D for herpes virus entry mediator [HVEM], a receptor expressed by T lymphocytes), whose potential involvement in MM is unknown. This evidence concerns the gene TNFSF14 and Miyoshi myopathy.