The calcium chloride-induced AAA in rat only induces the formation of aneurysm, with no consideration of hyperlipidemia, gender, atherosclerosis, thrombus, rupture, and other complicated factors in human disease, but the aneurysm induced by AngII in ApoE−/− mice generally considers atherosclerosis, thrombus, hyperlipidemia, gender, and rupture, and it is more fit for studying the mechanisms and treatment of AAA [1, 2, 11]. Here, AGT is linked to triple-A syndrome.