Concerning the immune responses to respiratory viruses in a type 2 milieu in animal models of asthma, a recent study showed that MØs from OVA-treated mice expressed eotaxin-1/CCL11, IL-4, and IL-13 in response to RV1B infection ex vivo, as well as the MØ alternative activation markers arginase-1, chitinase-like 3, MØ galactose-type C-type lectin 2, and IL-10 [133]. The gene discussed is CCL11; the disease is infection.