The study disclosed divergent cellular production profiles and plasma levels in AE patient groups and controls; Th17-type IL-17A levels were similar in patients with progressive, stable, and cured E. multilocularis metacestode lesions, IL-17B increased in AE patients, while the Th17-type IL-17F production was highest in controls and depressed in all AE patient groups similarly [31]. The gene discussed is IL17A; the disease is acrodermatitis enteropathica.