IFNG and autoimmune disease: TEC H/P in IFN-γ−/− NOD.H-2h4 mice is an autoimmune disease, because T cells from IFN-γ−/− mice with severe TEC H/P transfer similar lesions to IFN-γ−/− NOD.H-2h4.SCID mice, and IFN-γ−/− SCID mice, which lack lymphocytes, and mice lacking T cells do not develop TEC H/P [2,10].