Furthermore, high-throughput analysis of more than 6,000 cDNA mouse sequences indicated the expression of UGT2A2, a splice variant of UGT2A1, in neonatal OM whose glucuronidation activity towards several different endo- and xenobiotic substrates was similar to that of UGT2A1 (Strausberg et al., 2002; Sneitz et al., 2009; Court et al., 2012). The gene discussed is UGT2A1; the disease is ocular melanoma.