Red blood cells (RBCs) are particularly dependent on G6PD, and thus, deficiency is commonly manifested clinically as hemolysis triggered by exogenous factors, such as infections, drugs (e.g., 8-aminoquinolines), and in some cases, fava beans and selected other foods.6 G6PD deficiency occurs as a result of gene mutations located in the X chromosome. The gene discussed is G6PD; the disease is hyperinsulinemic hypoglycemia, familial, 4.