To determine if blockade of CD1d in the context of radiotherapy and CD137 costimulation resulted in improved priming of anti-tumor CD8+ T cells in dLN of WT mice similar to that observed during anti-CTLA-4 blockade (Figure 3), we measured tumor antigen-specific production of IFNγ by dLN cells. Here, TNFRSF9 is linked to neoplasm.