Thereby, we analyzed differential expression of ncRNAs in knockout mouse models for one of the two brain L-type calcium channels (Cav1.3) being implicated in Parkinson's disease (Sulzer and Surmeier 2013) and in lethargic mice, which harbor a nonsense mutation in the gene encoding for the auxiliary voltage-dependent Ca2+ channel β4 subunit (Cacnb4), previously described as a model for idiopathic epilepsy and ataxia (Burgess et al. 1997; Tadmouri et al. 2012). Here, CACNA1D is linked to Parkinson disease.