The remaining glucose uptake was likely mediated through other glucose transporters expressed by B-ALL cells.26, 27, 28 In particular, Gluts 3 and 6 may increase activity to support this glucose transport.17 Human B-ALL cells expressed additional transporters, including Glut4 and Glut5 (Supplementary Figure S1B). Here, SLC2A3 is linked to precursor B-cell acute lymphoblastic leukemia.