SLC8A1 and myocardial infarction: Manipulation of iPSC with lentivirus vectors encoding markers under the control of NCX1 promoter yields a large number of highly pure CM, and grafting of iPSC-derived CM on cell sheets over scarred areas of myocardium subsequent to coronary artery ligation reduced ventricular remodeling and LV fibrosis subsequent to new CM populations and new blood vessel formation, leading to enhanced restoration of cardiac function after MI.