Dysbindin-1 is expressed ubiquitously in the body and brain [1], and has been postulated to be implicated not only in psychiatric disorders such as schizophrenia, bipolar disorder, and METH psychosis [5,10-12,15-17], but also peripheral diseases such as type 7 Hermansky-Pudlak syndrome, which is accompanied with oculocutaneous albinism, prolonged bleeding, and pulmonary fibrosis due to abnormal vesicle trafficking to lysosomes and related organelles [2]. This evidence concerns the gene DTNBP1 and oculocutaneous albinism.