Although none of the molecular groups was significantly linked to the configuration of resection margin or inflammatory antitumor reaction in TNBC, our data suggest that PI3K driven tumors tended towards a more infiltrative phenotype (p=0.077), while TP53-driven neoplasms were by trend associated with stronger inflammation (p=0.099)(Table 1). The gene discussed is PIK3CG; the disease is neoplasm.