The authors thank Pierre Daublain for dinaciclib formulation optimization; Karen Leander, Kathleen Haskell, Traci Lifsted and Sharon Adamski for assistance with tumor resections; WuXi App Tec and Piedmont Research Laboratories for animal studies; Life Technologies - Oncomine Power Tools (Life Technologies, Ann Arbor, MI) for access to MCL1 amplification human tumor data; the Broad-Novartis Cancer Cell Line Encyclopedia (CCLE) Project for public access to cancer cell gene expression and genomics data and bear no responsibility for the further analysis or interpretation of their data. The gene discussed is MCL1; the disease is cancer.