The observations have highlighted possible roles for many factors, including platelet-activating factor receptor [8], urokinase receptor [9], ICAM-1 [10, 11], CD40 [12], neutrophils [13], vascular endothelial growth factor (VEGF) [14], epithelium sodium channel activity [15], CD36-dependent parasite sequestration [16], hemozoin deposition [17], and CD8+ T lymphocytes [18], in malaria-associated lung injury. This evidence concerns the gene VEGFA and malaria.