Tagashira et al.27 reported that IL-10 attenuates TNF-α-induced IL-6 synthesis via the NF-κB and MAPK pathways in endometriotic stromal cells, which suggests that increased IL-10 expression may have a significant role in regulating the balance between complex pro- and anti-inflammatory behaviors in endometriosis. Here, NFKB1 is linked to endometriosis.