Our data are also supported by previously published studies by us [11], [13], [27] and by others [9], [10] showing deregulation of ANXA1 (in CFTR−/− mouse), of GRP78 for UPR activation (in both CFNP and NP), of PRDX6, PRDX2, CATB, HSPB1, PDIA3, ANXA1, VIME and EZRI (in CF cells). The gene discussed is HSPA5; the disease is cystic fibrosis.