Our data are also supported by previously published studies by us [11], [13], [27] and by others [9], [10] showing deregulation of ANXA1 (in CFTR−/− mouse), of GRP78 for UPR activation (in both CFNP and NP), of PRDX6, PRDX2, CATB, HSPB1, PDIA3, ANXA1, VIME and EZRI (in CF cells). This evidence concerns the gene CFTR and cystic fibrosis.