APOB and steatosis: The decrease in steatosis might also be ascribed to the nitro-oxidative stress, because in vitro exposition of hepatic proteins to peroxynitrite reduces the amount of ApoB100 and ApoB45 (Solís-Muñoz et al., 2011), two proteins involved in both the assembly of triglycerides into very-low-density lipoprotein (VLDL) and VLDL secretion (Goldberg and Ginsberg, 2006).