In contrast, Oguro et al reported that global ischemia selectively increases Cx32 protein expression in parvalbumin-positive inhibitory interneurons in CA1, and Cx32-null mice exhibit enhanced vulnerability to global ischemia-induced damage, which suggest Cx32 may play a critical role in protection and survival of CA1 interneurons after global ischemia [9]. Here, GJB1 is linked to ischemia.