SLC6A1 and Generalized non-motor (absence) seizure: This, together with the already described loss of function of GAT1 in thalamic astrocytes of genetic models of absence seizures [29] and the fact that GABA transporters affect astrocytic [Ca2+]i transients [55], clearly stresses the importance of this glial cell type in the generation of these non-convulsive seizures.