The literature shows that p16 INK4a and p14ARF are deleted in 80-90% of cases of MM.(36,37) Approximately 70% of all cases of epithelial MM and nearly 100% of allcases of biphasic or sarcomatoid MM show changes in p16INK4a and p14 ARF. The gene discussed is CDKN2A; the disease is Miyoshi myopathy.